Storrs Limbless


Origin & History: Found in Rhode Island Red flock. Prahlad, KV et al. 1979 J Exp Zool 209:427-434; received from Bitgood in late 1980's.

Breed: White Leghorn background

Symbol: ll

Characteristics: Autosomal recessive, embryonic lethal

Description: Homozygotes do not develop wings or legs and usually have short upper beaks.

No image available

Additional information: Outgrowth of limb buds is initiated at HH stage 17/18 in limbless (ll) embryos, but the apical ectodermal ridge (AER) doesn't form at stage 18 and mutant embryos are readily identifiable at stage 19/20. Thus limb bud initiation does not require an AER but without it limb outgrowth ceases by stage 20/21. Limb bud mesoderm shows massive necrosis by stage 22: buds degenerate and disappear by stage 24. The defect is localized in the ectoderm. (Limb Development and Regeneration, pp 33-43 1983: Development 104:361-367, 1988)
As in the case of normal limb buds deprived of their AER, explanted ll limb buds can undergo proliferation and directed outgrowth in the presence of exogenous FGF-2 or FGF-4. IGF-1 or insulin is more effective; in their presence, the thin apical ectoderm of mutant limb buds is induced to form thickened AER- like structures that can express high levels of Msx-2, a marker for AER function. (Development 122:1323-1330, 1996)
Other gene products also stimulate enhanced outgrowth of ll limb buds and expression of appropriate genes under various experimental conditions:

(Development 122:3851-3861, 1996): Implants of beads soaked with Fgf-8 - Shh
(Dev Biol 184:343-350, 1997): FGF-2 supplemented culture medium - syndecan-3

(Dev Biol 202:43-55, 1998): TGF-alpha or EGF supplemented serum-free defined medium - Msx2, Bmp-2, Wnt7a but not Fgf-8, Fgf-4, Bmp-4 or Shh

Expression of genes associated with initiation of limb bud outgrowth has been compared in ll and normal embryos for the following:

(Development 113:1487-1493, 1996): Ghox-7, Ghox-8

(Growth Hormone & IGF Res 11:346-363, 2001): IGF-1, IGFBP2

(Development 122:2319-2330, 1996): Fgf-2, Fgf-4, Fgf-8, Bmp-2, Bmp-4, Hoxd-11, Hoxd-12, Hoxd-13, Shh, Wnt7a, En-1, C-Lmx1

(Dev Biol 179:339-346, 1996): Fgf-4, Bmp-2, Bmp-4 Bmp-7, TCF-1, Msx-2, CEK-3, Hoxd-11, Hoxd-12, Hoxd-13

(Development 122:3851-3861, 1996): Fgf4, Fgf8, Shh, Hoxd-11, Hoxd-13, Wnt7a, Lmx1, En1

(Dev Biol 184:343-350, 1997): syndecan-3

(Dev Biol 202:43-55, 1998): TGF-alpha, EGF, c-erb B

(Dev Genet 25:331-338, 1999): Fgf-10, FGFR2b, Fgf-8, FGFR2c

(Develop. Growth Differ. 44:315-325, 2002): Wnt3a, Frizzled-1, Frizzled-7, Frizzled-3, Frizzled-6, Lef-1, Tcf-1, Axin, B-catenin



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